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Question

目的：探讨肝硬化自发性腹膜炎的临床特点及有关诱因与治疗措施。方法：对我院消化内科2004年1月至2008年1月住院的肝硬化并发自发性腹膜炎患者62例的临床资料进回顾性分析。结果：肝硬化并发自发性腹膜炎的发生率为9。09%，致病菌G-菌多见。结论：肝硬化并发自发性腹膜炎临床特点起病不典型，大多数患者感染中毒症状不明显，腹膜炎体征不典型。以腹胀明显加重，腹水短期增多，利尿治疗效果差为主要表现。腹水细胞记数和细菌培养是自发性腹膜炎（SBP）的诊断依据。肝硬化自发性腹膜炎可以通过综合支持治疗，适当应用抗生素治疗而得到预防。

Answer 1

Goal: Discussion liver cirrhosis spontaneous peritonitis's clinical characteristic and related cause and remedial measure. Method: Was hospitalized January, 2004 the liver cirrhosis concurrent spontaneous peritonitis patient 62 example clinical material which to January, 2008 enters the review analysis to my courtyard digestion internal medicine department. Finally: Liver cirrhosis concurrent spontaneous peritonitis's formation rate is 9. 09%, the pathogenic bacteria G- fungus sees. Conclusion: The liver cirrhosis concurrent spontaneous peritonitis clinical characteristic gets up gets sick not typically, the majority patients infect the symptom of poisoning not to be obvious, the peritonitis symptom is atypical. By the distension of the abdomen obvious aggravation, the ascites short-term increases, the diuresis treatment result difference is the main performance. The ascites cell register and the bacilliculture are the spontaneous peritonitis (SBP) diagnosis basis. The liver cirrhosis spontaneous peritonitis may through the synthesis support treatment, obtain the prevention suitably using the antibiotic treatment.

Objective: To investigate the spontaneous peritonitis cirrhosis of the clinical features and treatment measures and incentives. Method: digestive internal medicine from January 2004 to January 2008 hospitalized patients with liver cirrhosis complicated by spontaneous peritonitis 62 cases of clinical data into the retrospective analysis. Results: cirrhosis spontaneous peritonitis complicated by the occurrence rate of 9.09 percent, the G-pathogenic bacteria found. Conclusion: cirrhosis complicated by spontaneous peritonitis is not a typical clinical features of onset, the majority of infections in patients with obvious symptoms of poisoning, not the typical signs of peritonitis. In order to significantly increase the abdominal distension, ascites short-term increase in inappropriate treatment for the poor performance of the principal. Ascites cell count and culture is spontaneous bacterial peritonitis (SBP) on the basis of the diagnosis. Cirrhosis spontaneous peritonitis can be integrated to support the treatment, the proper use of antibiotics in the treatment and prevention.

Answer 2

Goal: Discussion liver cirrhosis spontaneous peritonitis's clinical characteristic and related cause and remedial measure. Method: Was hospitalized January, 2004 the liver cirrhosis concurrent spontaneous peritonitis patient 62 example clinical material which to January, 2008 enters the review analysis to my courtyard digestion internal medicine department. Finally: Liver cirrhosis concurrent spontaneous peritonitis's formation rate is 9. 09%, the pathogenic bacteria G- fungus sees. Conclusion: The liver cirrhosis concurrent spontaneous peritonitis clinical characteristic gets up gets sick not typically, the majority patients infect the symptom of poisoning not to be obvious, the peritonitis symptom is atypical. By the distension of the abdomen obvious aggravation, the ascites short-term increases, the diuresis treatment result difference is the main performance. The ascites cell register and the bacilliculture are the spontaneous peritonitis (SBP) diagnosis basis. The liver cirrhosis spontaneous peritonitis may through the synthesis support treatment, obtain the prevention suitably using the antibiotic treatment.本回答被提问者采纳

Answer 3

Objective: To investigate the spontaneous peritonitis cirrhosis of the clinical features and treatment measures and incentives. Method: digestive internal medicine from January 2004 to January 2008 hospitalized patients with liver cirrhosis complicated by spontaneous peritonitis 62 cases of clinical data into the retrospective analysis. Results: cirrhosis spontaneous peritonitis complicated by the occurrence rate of 9.09 percent, the G-pathogenic bacteria found. Conclusion: cirrhosis complicated by spontaneous peritonitis is not a typical clinical features of onset, the majority of infections in patients with obvious symptoms of poisoning, not the typical signs of peritonitis. In order to significantly increase the abdominal distension, ascites short-term increase in inappropriate treatment for the poor performance of the principal. Ascites cell count and culture is spontaneous bacterial peritonitis (SBP) on the basis of the diagnosis. Cirrhosis spontaneous peritonitis can be integrated to support the treatment, the proper use of antibiotics in the treatment and prevention.